Search results for "Glycine Receptor"

showing 10 items of 20 documents

Putative Role of Taurine as Neurotransmitter During Perinatal Cortical Development

2017

Neurotransmitters and neuronal activity affect neurodevelopmental events like neurogenesis, neuronal migration, apoptosis and differentiation. Beside glutamate and gamma-amino butyric acid, the aminosulfonic acid taurine has been considered as possible neurotransmitter that influences early neuronal development. In this article I review recent studies of our group which demonstrate that taurine can affect a variety of identified neuronal populations in the immature neocortex and directly modulates neuronal activity. These experiments revealed that taurine evoke dose-dependent membrane responses in a variety of neocortical neuron populations, including Cajal-Retzius cells, subplate neurons a…

0301 basic medicineTaurineNeocortexGlutamate receptor03 medical and health scienceschemistry.chemical_compound030104 developmental biology0302 clinical medicinemedicine.anatomical_structurenervous systemchemistryPostsynaptic potentialmedicineGABAergicPremovement neuronal activityNeuronGlycine receptorNeuroscience030217 neurology & neurosurgery
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ChemInform Abstract: Benzimidazoles as NMDA Glycine-Site Antagonists: Study on the Structural Requirements in 2-Position of the Ligand.

2010

A series of different substituted benzimidazole derivatives has been synthesized and evaluated for the ability to displace [3H]MDL-105,519 to rat cortical membranes. Two benzimidazole-2-carboxylic acids 9 b and 9 c, in this substitution pattern not yet described as glycine antagonists, showed IC50 values of 0.89 microM (9 b) and 38.0 microM (9 c). Replacement of the carboxylate function in 2-position by a sulfonic acid moiety appreciably increased solubility, but decreased the affinity giving evidence for the strong need of the carboxylate group within the ligand. Further structure-activity studies using benzimidazole-2-one derivatives with an acetic acid moiety adjacent to a ring nitrogen …

chemistry.chemical_classificationBenzimidazolechemistry.chemical_compoundChemistryStereochemistryLigandAmideGlycineMoietyGeneral MedicineGlycine receptor antagonistCarboxylateSulfonic acidChemInform
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A Novel Series of 2-Carboxytetrahydroquinolines Provides New Insights into the Eastern Region of Glycine Site NMDA Antagonists

2000

A series of potent 4-substituted tetrahydroquinolines has been synthesized and biologically tested in order to refine the eastern region of the pharmacophore model for glycine site NMDA antagonists concerning the assessment of lipophilicity, flexibility, and hydrogen bonding. Displacement studies on rat cortical membranes using [ 3 H]-5,7-dichlorokynurenic acid as a radioligand indicated that binding affinities are markedly enhanced when additional hydrogen-accepting groups are introduced into the eastern region of the 2-carboxytetrahydroquinolines. Among the most potent ligands were some urea, sulfonylurea, and crown ether compounds as interesting leads for new diagnostics, especially for …

chemistry.chemical_classificationBiodistributionChemistryStereochemistryDrug DiscoveryGlycineLipophilicityRadioligandPharmaceutical ScienceNMDA receptorPharmacophoreGlycine receptorCrown etherArchiv der Pharmazie
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Hyperekplexia caused by dominant-negative suppression of glyra1 function.

2007

Hyperekplexia (HE; startle disease; OMIM#149400) is a rare inheritable neurologic disorder characterized by an exaggerated response to sudden stimuli, muscular rigidity, and hyperreflexia, leading to chronic injuries due to unprotected falls. All symptoms are present at birth but gradually decline during the first year of life, although an exaggerated startle response remains during adulthood.1 Dysfunctional inhibitory neurotransmission by glycine (Gly) plays a central role in HE pathogenesis. All patients with HE carry mutations in genes encoding either for α1 (GLYRA1) or β (GLYRB) Gly receptor subunits, presynaptic Gly transporters (SLC6A5), or proteins involved in Gly receptor (GLYR) clu…

Malemedicine.medical_specialtySubunitReflex StartleNonsense mutationCompound heterozygosityGeneReceptors GlycineInternal medicinemedicineMissense mutationHumansGlycine ReceptorHyperekplexiaGlycine receptorNervous System DiseaseGeneticsStartle DiseaseNeuroscience (all)GephyrinbiologyInfantPenetrancePedigreeEndocrinologyHyperekplexiaNON PREVISTO DA NORME REDAZIONALI (“NEUROLOGY”)Codon NonsenseMutationbiology.proteinNeurology (clinical)medicine.symptomNervous System DiseasesCollybistinHuman
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Activation of glycine receptors modulates spontaneous epileptiform activity in the immature rat hippocampus

2014

While the expression of glycine receptors in the immature hippocampus has been shown, no information about the role of glycine receptors in controlling the excitability in the immature CNS is available. Therefore, we examined the effect of glycinergic agonists and antagonists in the CA3 region of an intact corticohippocampal preparation of the immature (postnatal days 4-7) rat using field potential recordings. Bath application of 100 μM taurine or 10 μM glycine enhanced the occurrence of recurrent epileptiform activity induced by 20 μM 4-aminopyridine in low Mg(2+) solution. This proconvulsive effect was prevented by 3 μM strychnine or after incubation with the loop diuretic bumetanide (10 …

TaurinePhysiologyTaurineGlycinePharmacologyHippocampuschemistry.chemical_compoundNeuroscience: Development/Plasticity/RepairReceptors GlycinemedicineAnimalsRats WistarReceptorGlycine receptorCells CulturedEpilepsyChemistryGABAA receptorStrychnineRatsBiochemistrynervous systemAnimals NewbornGlycineGabazineAnticonvulsantsBumetanidemedicine.drug
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Glycine receptors influence radial migration in the embryonic mouse neocortex.

2011

To investigate whether glycine receptors influence radial migration in the neocortex, we analyzed the effect of glycine and the glycinergic antagonist strychnine, on the distribution of 5-bromo-2'deoxyuridine-labeled neurons in organotypic slice cultures from embryonic mice cortices. Application of glycine impeded radial migration only in the presence of the glycine-transport blockers, ALX-5407 and ALX-1393. This effect was blocked by the specific glycine receptor antagonist strychnine, whereas application of strychnine in the absence of glycine was without effect. We conclude from these observations that an activation of glycine receptors can impede radial migration, but that the glycinerg…

GlycineCell CountNeocortexBiologychemistry.chemical_compoundMiceOrgan Culture TechniquesReceptors GlycineCell MovementGlial Fibrillary Acidic ProteinmedicineAnimalsDrug InteractionsReceptorGlycine receptorNeuronsNeocortexGeneral NeuroscienceAntagonistGlycine AgentsSarcosineGlycine receptor antagonistStrychnineStrychnineEmbryo MammalianCell biologymedicine.anatomical_structurechemistryBromodeoxyuridineCerebral cortexPhosphopyruvate HydrataseGlycineNeuroscienceNeuroreport
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Antagonists and agonists at the glycine site of the NMDA receptor for therapeutic interventions.

2003

For decades neuroreceptor research has focused on the development of NMDA glycine-site antagonists, after Johnson and Ascher found out in 1987 about the co-agonistic character of this achiral amino acid at the NMDA receptor. Contrary to the inhibitory glycine receptor (glycine(A)) the glycine binding site on the NMDA receptor (glycine(B)) is strychnine-insensitive. A great diversity of diseases showing a disturbed glutamate neurotransmission have been linked to the NMDA receptor. Glycine site antagonists have been investigated for acute diseases like stroke and head trauma as well as chronic ones like dementia and chronic pain.

PsychosisGlycinePainPharmacologyNeurotransmissionInhibitory postsynaptic potentialReceptors N-Methyl-D-AspartateGlycine bindingMemantineDrug DiscoverymedicineAnimalsHumansReceptorGlycine receptorPharmacologychemistry.chemical_classificationBinding SitesEpilepsyOrganic ChemistryGlutamate receptorGeneral Medicinemedicine.diseaseAmino acidStrokeNeuroprotective AgentsBiochemistrychemistryGlycineSchizophreniaNMDA receptorAnticonvulsantsDementiaExcitatory Amino Acid AntagonistsEuropean journal of medicinal chemistry
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Benzimidazoles as NMDA Glycine-Site Antagonists: Study on the Structural Requirements in 2-Position of the Ligand

2000

A series of different substituted benzimidazole derivatives has been synthesized and evaluated for the ability to displace [3H]MDL-105,519 to rat cortical membranes. Two benzimidazole-2-carboxylic acids 9 b and 9 c, in this substitution pattern not yet described as glycine antagonists, showed IC50 values of 0.89 microM (9 b) and 38.0 microM (9 c). Replacement of the carboxylate function in 2-position by a sulfonic acid moiety appreciably increased solubility, but decreased the affinity giving evidence for the strong need of the carboxylate group within the ligand. Further structure-activity studies using benzimidazole-2-one derivatives with an acetic acid moiety adjacent to a ring nitrogen …

chemistry.chemical_classificationBenzimidazoleChemistryStereochemistryLigandPharmaceutical ScienceGlycine receptor antagonistSulfonic acidchemistry.chemical_compoundAmideDrug DiscoveryGlycineMoietyCarboxylateArchiv der Pharmazie
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Stereospecific interaction of bicuculline with specific [3H]strychnine binding to rat spinal cord synaptosomal membranes

1980

The gamma-aminobutyric acid (GABA) antagonist (+/-)-bicuculline inhibits specific [3H]strychnine binding to postsynaptic glycine receptor sites in rat spinal cord synaptosomal membranes with an inhibition constant of about 5 microM, which is fairly similar to its inhibition constant reported for the GABA receptor. This effect is highly stereospecific, since the affinity of (-)-bicuculline for the specific [3H]strychnine binding sites is more than ten times less than that of the pharmacologically active (+)-bicuculline. Besides an unspecific effect at the glycine receptor, the results could suggest that the glycine and the GABA receptors are located close together in spinal cord membranes, s…

GlycineSynaptic MembranesReceptors Cell SurfaceBicucullineBinding CompetitiveStructure-Activity Relationshipchemistry.chemical_compoundReceptors GlycineSpecies SpecificityGABA receptorPostsynaptic potentialmedicineAnimalsReceptorGlycine receptorChemistryGABAA receptorGeneral NeuroscienceStereoisomerismStrychnineStrychnineBicucullineRatsSpinal CordBiochemistryGlycineBiophysicsmedicine.drugNeuroscience Letters
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Hydantoin-substituted 4,6-dichloroindole-2-carboxylic acids as ligands with high affinity for the glycine binding site of the NMDA receptor.

2002

A novel series of C-3 substituted 4,6-dichloroindole-2-carboxylic acids was synthesized to investigate the influence of different hydrogen-bond donor and acceptor groups at this specific position on the affinity to the glycine site of the NMDA receptor. These novel 3-indolylmethyl derivatives with ring-open (amines, sulfonamides, amides, ureas) and cyclic substituents (imidazolidin-2-ones, (thio)hydantoins) led to the discovery that compounds bearing a hydantoin substituent at the C-3 position of the indole nucleus are the most promising ones. In this series the hydantoins, ureas, and imidazolidin-2-ones were identified as very potent inhibitors of the binding of the glycine site specific l…

IndolesStereochemistrySwineGlycineHydantoinThio-In Vitro TechniquesLigandsBinding CompetitiveReceptors N-Methyl-D-Aspartatechemistry.chemical_compoundMiceRadioligand AssayStructure-Activity RelationshipGlycine bindingSeizuresDrug DiscoveryAnimalsBinding siteGlycine receptorIndole testElectroshockBinding SitesBicyclic moleculeHydantoinsBrainRatschemistryGlycineMolecular MedicineAnticonvulsantsFemaleJournal of medicinal chemistry
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